Oxalacetic acid (OAA) Oxalacetate highest purity at wholesale price 1kg/bag

Oxalacetic acid (OAA) Oxalacetate highest purity at wholesale price 1kg/bag

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Oxalacetic acid,(oxaloacetate ) highest purity, at wholesale price, 1kg/bag, promotion price end in 30th, June. 2016

Other name: 2-ketosuccinic acid, 2-oxobutanedioate , 2-oxobutanedioic acid , 2-oxobutanedioic acid ketosuccinic acid , 2-oxosuccinic acid, 3-carboxy-3-oxopropanoic acid, Alpha-ketosuccinic acid , Butanedioic acid, oxo- , Butanedioic acid, oxo- (9CI) , Butanedioic acid, oxo-, disodium salt ,C00036 , Keto-oxaloacetate , Ketosuccinic acid ,OAA , Oxalacetate ,,Oxalacetic Acid ,Oxalacetic acid (8CI) , Oxalacetic acid, disodium salt , Oxaloacetate , Oxaloacetate Ion , Oxaloacetate(2-) , Oxaloacetic acid ,Oxaloethanoic acid , Oxobutanedioate,

CAS Number:328-42-7

Chemical & Physical Property
Appearance: white powder or almost white crystalline
Assay(Titration): 95-102%
Melting Point: about 161degree
Related Substances: 0.5% max.
Residue on Ignition: 0.5% max.
Total Volatiles: 0.5% max.
Sulfate: 20ppm max.
Chloride: 5ppm max.
Heavy Metals(Pb): 10ppm max.
Total Plate Count: 1000cfu/gram max.
Yeast & Mold: 100cfu/gram max.
E.coli: Negative
Salmonella: Negative
Shelf Life: 24months

Index source:


1. Oxaloacetate increases energy production.
Cells can produce energy in two different ways. The more primitive, less effective method is glycolysis. The more advanced and efficient method is oxidative phosphorylation. This occurs in the mitochondria, and it produces eight times more energy than glycolysis-while using the same amount of fuel.[1]
Oxaloacetate is an important part of the metabolic cycle that allows oxidative phosphorylation to occur. Without this molecule, cells are forced to rely more heavily on glycolysis.[2] This can lead to decreased production of energy and a decreased supply of blood sugar. In fact, studies indicate that a lack of oxaloacetate can cause energy production to fall by nearly 500%.[3]

2. Oxaloacetate starves cancer.
Cancerous cells rely on simple sugars and glutamate for their fuel supply, and this is especially true for brain tumors.[4] Glutamate also allows cancer cells to grow and divide.[5] When simple sugars and glutamate are abundant, cancer cells can produce more energy. This makes them more capable of spreading and more resistant to treatment.
Fortunately, studies have shown that the consumption of an oxaloacetate supplement can decrease blood glutamate levels by 40%.[6,7] This leads to a reduction of tumor size and invasiveness.[8] Furthermore, patients who are given an oxaloacetate supplement experience an increased survival rate of 237%.[8]

3. Oxaloacetate reduces brain trauma.

In addition to its use as a cancer fuel, glutamate can also trigger the excitation of neurons.[9] This is especially dangerous to stroke victims, and over time, it causes damage to brain cells. However, as oxaloacetate removes glutamate from the bloodstream, glutamate in the brain and spinal fluid is forced to exit, as well.[10]

This allows the brain to recover from strokes and traumatic injuries.[10] Without glutamate, neurons can naturally restore their long-term potentiation, allowing the cells to transmit signals more quickly, which leads to faster neural communication and improved brain function.

How to find an oxaloacetate supplement

Because oxaloacetate is a cellular metabolite, there is no effective way to obtain it from dietary sources. It must be refined and used as a supplement, and is available in capsule form. As a natural part of the body's cellular processes, oxaloacetate is a safe supplement with no side effects. Read about another glutamate-fighting supplement, N-acetyl cysteine here.


[1] The Cell: A Molecular Approach. 2nd edition.

[2] Biochemistry. 5th edition.

[3] J Neurochem. 1988 Mar;50(3):673-80.

[4] Invest New Drugs. 2012 Feb 2.

[5] Trends Biochem Sci. Aug 2012; 35(8): 427-433.

[6] Exp Neurol. 2007 Jan;203(1):213-20.

[7] Neurotherapeutics. July 2012; 9(3): 649-657.

[8] Invest New Drugs. 2012 Feb 2.

[9] Stroke. 1996 Jun;27(6):1060-5.

[10] Eur J Pharmacol. 2009 Feb 14;604(1-3):51-7.

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